(Solved): please help ASAP!! will rate Chapter 11 Assignment 4 Zuri has returned home from a semester in Ethio ...

Chapter 11 Assignment 4 Zuri has returned home from a semester in Ethiopia. After 20 hours of traveling, Zuri was tired. She complained of dizziness and nausea and was warm to the touch. She barely made it to the bathroom before vomiting. Her mom found her trembling with chills. She has a history of atrial fibrillation and when she ran out of medicine while in Ethiopia, took an herbal remedy for it. She took several doses while traveling because of her anxiety. While conversing with her mom, she had a mild seizure and was rushed to the hospital. I. List Zuri's signs and symptoms and any important notes: 2. What potential diagnoses are you considering? At the hospital, her vitals were taken: 3. What new signs and symptoms is Zuri experiencing? 4. What changes do you want to make to your potential diagnoses? 5. Use the Nernst equation (below) to calculate the equilibrium potential for potassium in a normal patient and in Zuri. Assume that the ion level in the interstitial fluid is the same as that in the blood plasma, and also assume that the intracellular concentration of potassium is in Zuri and in normal patients. (Don't be intimidated by the formula, just plug what you know into the formula using a calculator.) 6. The Nernst equation is not measuring RMP, just the potential exhibited by potassium. However, if the resting membrane potential exhibits the same change as the equilibrium potential for potassium we can predict how Zuri's ion imbalance will affect a cell's resting membrane potential. To do this, you need to compare Zuri's cquilibrium potential for potassium to the normal equilibrium potential for potassium. What is the difference between Zuri and normal'? (Use the Nernst equation to calculate the normal potassium values provided in the second table.) 7. If Zuri's RMP has the same difference, would Zuri's resting membrane potential be hyperpolarized or depolarized? 8. If the value for threshold in Zuri is numerically identical to that of a normal patient, what would be the effect of Zuri's change in resting membrane potential on the amount of depolarization required to produce an action potential? 9. Can this change (the difference between the resting membrane potential and threshold) explain any of Zuri's symptoms? If so, state which ones and explain why. It turns out the herbal remedy contained a drug called Digoxin, which is an extract from plants like the foxglove. Na/K-ATPase pumps located in your cell membranes control the concentration of sodium and potassium ions inside and outside of your cells. These ion levels must remain reasonably constant because they are instrumental in producing the resting membrane potential of your cells. Excess Digoxin inhibits Na/K-ATPase pumps so that the membrane potentials become less negative and too many action potentials are produced. 10. How does the mechanism of Digoxin toxicity explain Zuri's signs and symptoms? 11. What would happen if a drug increased the activity of sodium/potassium pumps? 12. How can electrolyte imbalances cause problems in your muscles and neurons?

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